Filters

Marieluise Kirchner, Dieter Jenne, Lovis Kling, Claudia Eulenberg-Gustavus, Uwe Jerke, Maximilian Ebert, Philipp Mertins, Ralph Kettritz, Alan D Salama, Adrian Schreiber, Kai-Uwe Eckardt, Saban Elitok, Markus Bieringer

The Journal of clinical investigation 2022;132(23):e160089

Free Full Text

Abstract

Complement receptor Mac-1 is an adaptor for NB1 (CD177)-mediated PR3-ANCA neutrophil activation.

Ralph Kettritz, Friedrich Luft, Anje Sporbert, Sentot Santoso, Behnaz Bayat, Gunnar Dittmar, Susanne Rolle, Uwe Jerke

The Journal of biological chemistry 2011;286(9):7070-81

Free Full Text

Abstract

Antineutrophil cytoplasmic antibodies (ANCAs) in patients with inflammatory bowel disease show no correlation with proteinase 3, lactoferrin, myeloperoxidase, elastase, cathepsin G and lysozyme: a Singapore study.

B L Lim, W K Cheong, A E Ling, H S Ng, C J Ooi

Annals of the Academy of Medicine, Singapore 2001;29(6):704-7

Full Text

Abstract

Immunotherapeutic strategies in chronic myeloid leukemia.

Richard E Clark

Current hematologic malignancy reports 2010;2(2):89-94

Full Text

Abstract

Disease Activity, Antineutrophil Cytoplasmic Antibody Type, and Lipid Levels in Antineutrophil Cytoplasmic Antibody-Associated Vasculitis.

E William St Clair, John H Stone, Hyon Choi, Yuqing Zhang, Robert Spiera, Ulrich Specks, Philip Seo, Paul Monach, Peter A Merkel, Carol A Langford, Cees G M Kallenberg, Katherine Liao, Xiaoqing Fu, Zachary S Wallace

Arthritis & rheumatology (Hoboken, N.J.) 2020;71(11):1879-1887

Free Full Text

Abstract

Priming by tumor necrosis factor-alpha of human neutrophil NADPH-oxidase activity induced by anti-proteinase-3 or anti-myeloperoxidase antibodies.

Dominique Reumaux, Peter L Hordijk, Patrick Duthilleul, Dirk Roos

Journal of leukocyte biology 2007;80(6):1424-33

Full Text

Abstract

Results: 1 – 13 / 13

Filters

Expert Opinion

Text Availability

Publication Types

Publication Year

Results for ""Myeloblastin""[mesh_terms]

The neutrophil serine protease PR3 induces shape change of platelets via the Rho/Rho kinase and Ca(2+) signaling pathways.

Neutrophil elastase, proteinase 3 and cathepsin G: physicochemical properties, activity and physiopathological functions.

Human Proteinase 3, an important autoantigen of c-ANCA associated vasculitis, shares cross-reactive epitopes with serine protease allergens from mites: an analysis.

Competitively disrupting the neutrophil-specific receptor-autoantigen CD177:proteinase 3 membrane complex reduces anti-PR3 antibody-induced neutrophil activation.

Proteinase-3 directly activates MMP-2 and degrades gelatin and Matrigel; differential inhibition by (-)epigallocatechin-3-gallate.

B cell epitope specificity in ANCA-associated vasculitis: does it matter?

New insights that link microbes with the generation of antineutrophil cytoplasmic autoantibodies: the theory of autoantigen complementarity.

Protective α1-antitrypsin effects in autoimmune vasculitis are compromised by methionine oxidation.

Complement receptor Mac-1 is an adaptor for NB1 (CD177)-mediated PR3-ANCA neutrophil activation.

Antineutrophil cytoplasmic antibodies (ANCAs) in patients with inflammatory bowel disease show no correlation with proteinase 3, lactoferrin, myeloperoxidase, elastase, cathepsin G and lysozyme: a Singapore study.

Immunotherapeutic strategies in chronic myeloid leukemia.

Disease Activity, Antineutrophil Cytoplasmic Antibody Type, and Lipid Levels in Antineutrophil Cytoplasmic Antibody-Associated Vasculitis.

Priming by tumor necrosis factor-alpha of human neutrophil NADPH-oxidase activity induced by anti-proteinase-3 or anti-myeloperoxidase antibodies.

Results for
""Myeloblastin""[mesh_terms]