Efficacy and Safety of Alirocumab in Children and Adolescents With Homozygous Familial Hypercholesterolemia: Phase 3, Multinational Open-Label Study.

Marie T Baccara-Dinet, Eric Bruckert, Sonia Caprio, Albert Wiegman, Min-Ji Charng, Cézar A Zárate-Morales, Garen Manvelian, Anne Ourliac, Michel Scemama, Stephen R Daniels

Journal: Arteriosclerosis, thrombosis, and vascular biology 2022;42(12):1447-1457

PMID: 36325897

Abstract

BACKGROUND

Despite progress in treating homozygous familial hypercholesterolemia, most patients do not achieve low-density lipoprotein cholesterol (LDL-C) targets. This study examined efficacy and safety of the PCSK9 (proprotein convertase subtilisin/kexin type 9) inhibitor, alirocumab, in pediatric patients (aged 8-17 years) with inadequately controlled homozygous familial hypercholesterolemia.

METHODS

In this open-label, single-arm, multinational, Phase 3 study, patients (n=18) received alirocumab 75 mg or 150 mg (bodyweight <50 kg/≥50 kg) every 2 weeks as an adjunct to background treatment. The primary endpoint was percent change in LDL-C from baseline to Week 12. Secondary endpoints included changes in LDL-C and other lipid parameters up to 48 weeks, safety/tolerability, and alirocumab pharmacokinetics.

RESULTS

The mean age of patients was 12.4 years; 16/18 (89%) had mutations in the low-density lipoprotein receptor gene ) and 2/18 (11%) had mutations in the LDLR adapter protein 1 gene ( At baseline, mean LDL-C (standard deviation) was 373.0 (193.5) mg/dL, which decreased by 4.1% at Week 12 (primary endpoint) and 11.4%, 13.2%, and 0.4% at Weeks 4, 24, and 48, respectively. At Week 12, 9/18 (50%) patients achieved LDL-C reductions ≥15%. Mean absolute LDL-C decreases ranged from 25 to 52 mg/dL over follow-up. A post hoc analysis demonstrated heterogeneity of responses according to genotype. There were no unexpected safety/tolerability findings. Free PCSK9 was reduced to near zero for all patients at Weeks 12 and 24.

CONCLUSIONS

The study supports the efficacy and safety of alirocumab as a potential adjunct to treatment for some pediatric patients with homozygous familial hypercholesterolemia.

REGISTRATION

URL: https://www.

CLINICALTRIALS

gov; NCT03510715.

Address: Hôpital Pitié Salpêtrière, Sorbonne University Paris, France (E.B.).; Yale Pediatric Endocrinology, New Haven, CT (S.C.).; Department of Metabolic Disorders, Amsterdam UMC, Location AMC, Emma Children's Hospital, the Netherlands (A.W.).; Division of Cardiology, Department of Medicine, Taipei Veterans General Hospital, Taiwan, R.O.C. (M.-J.C.).; School of Medicine, National Yang Ming Chiao Tung University, Taipei, Taiwan, R.O.C. (M.-J.C.).; Hospital "Presidente Juárez" del Instituto de Seguridad y Servicios Sociales de los Trabajadores del Estado (ISSSTE), Oaxaca, Oax, México (C.A.Z.-M.).; Sanofi, Montpellier, France (M.T.B.-D.).; Regeneron Pharmaceuticals Inc, Tarrytown, NY (G.M.).; Aixial, Boulogne-Billancourt, France (A.O.).; Sanofi, Chilly-Mazarin, France (M.S.).; University of Colorado School of Medicine, Aurora (S.R.D.).
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