IGF-1 and IGFBP-3 in Inflammatory Cachexia.

Ana Isabel Martín, Teresa Priego, Álvaro Moreno-Ruperez, Daniel González-Hedström, Miriam Granado, Asunción López-Calderón

Journal: International journal of molecular sciences 2021;22(17):9469

PMID: 34502376

Abstract

Inflammation induces a wide response of the neuroendocrine system, which leads to modifications in all the endocrine axes. The hypothalamic-growth hormone (GH)-insulin-like growth factor-1 (IGF-1) axis is deeply affected by inflammation, its response being characterized by GH resistance and a decrease in circulating levels of IGF-1. The endocrine and metabolic responses to inflammation allow the organism to survive. However, in chronic inflammatory conditions, the inhibition of the hypothalamic-GH-IGF-1 axis contributes to the catabolic process, with skeletal muscle atrophy and cachexia. Here, we review the changes in pituitary GH secretion, IGF-1, and IGF-1 binding protein-3 (IGFBP-3), as well as the mechanism that mediated those responses. The contribution of GH and IGF-1 to muscle wasting during inflammation has also been analyzed.

Address: Department of Physiology, Faculty of Medicine, Complutense University of Madrid, 28040 Madrid, Spain.; Department of Physiology, Faculty of Nursing, Physiotherapy and Podiatry, Complutense University of Madrid, 28040 Madrid, Spain.; Department of Physiology, Faculty of Medicine, Autonomous University of Madrid, 28049 Madrid, Spain.; Pharmactive Biotech Products S.L. Parque Científico de Madrid, Avenida del Doctor Severo Ochoa, 37 Local 4J, 28108 Alcobendas, Spain.; Department of Physiology, Faculty of Medicine, Autonomous University of Madrid, 28049 Madrid, Spain.; CIBER Fisiopatología de la Obesidad y Nutrición, Instituto de Salud Carlos III, 28029 Madrid, Spain.
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