Is insulin an antiinflammatory molecule?

U N Das

Journal: Nutrition (Burbank, Los Angeles County, Calif.) 2002;17(5):409-13

PMID: 11377135

Full Text on Elsevier Science View on PubMed

Abstract

I suggest that insulin suppresses the secretion and antagonizes the harmful effects of tumor necrosis factor-alpha, macrophage migration-inhibitory factor, and superoxide anion. Therefore, the glucose-insulin-potassium regimen might be beneficial in acute myocardial infarction and useful in the management of patients with septicemia, septic shock, and other inflammatory diseases in which tumor necrosis factor-alpha and macrophage migration-inhibitory factor have important roles.

Address: From EFA Sciences LLC, Norwood, Massachusetts, USA. [email protected]

DOI: 10.1016/s0899-9007(01)00518-4

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MeSH Terms: Diabetic Ketoacidosis, Fatty Acids, Essential, Glucose, Humans, Insulin, Insulin Resistance, Macrophage Migration-Inhibitory Factors, Models, Biological, Myocardial Infarction, Potassium, Sepsis, Shock, Septic, Superoxides, Tumor Necrosis Factor-alpha