Selenium rescues orbital fibroblasts from cell death induced by hydrogen peroxide: another molecular basis for the effects of selenium in graves' orbitopathy.

Giovanna Rotondo Dottore, Riccardo Chiarini, Maria De Gregorio, Marenza Leo, Giamberto Casini, Luca Cestari, Stefano Sellari-Franceschini, Marco Nardi, Paolo Vitti, Claudio Marcocci, Michele Marinò

Journal: Endocrine 2018;58(2):386-389

PMID: 28097622

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Abstract

Address: Department of Clinical and Experimental Medicine, Endocrinology Units, University of Pisa and University Hospital of Pisa, Via Paradisa 2, Pisa, 56124, Italy.; Department of Surgical, Medical and Molecular Pathology, Ophthalmopathy Unit I, University of Pisa and University Hospital of Pisa, Via Paradisa 2, Pisa, 56124, Italy.; Department of Surgical, Medical and Molecular Pathology, ENT Unit I, University of Pisa and University Hospital of Pisa, Italy, Via Paradisa 2, Pisa, 56124, Italy.; Department of Clinical and Experimental Medicine, Endocrinology Units, University of Pisa and University Hospital of Pisa, Via Paradisa 2, Pisa, 56124, Italy. [email protected].

DOI: 10.1007/s12020-016-1226-9

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https://dx.doi.org/10.1007/s12020-016-1226-9

MeSH Terms: Adipose Tissue, Aged, Cell Death, Cells, Cultured, Cytoprotection, Female, Fibroblasts, Graves Ophthalmopathy, Humans, Hydrogen Peroxide, Male, Middle Aged, Orbit, Primary Cell Culture, Selenium, Signal Transduction