Understanding the interaction between environmental exposures and molecular events in colorectal carcinogenesis.

N Ishibe, A N Freedman

Journal: Cancer investigation 2001;19(5):524-39

PMID: 11458819

Abstract

Address: Genetic Epidemiology Branch, Division of Cancer Epidemiology and Genetics, National Cancer Institute, 6120 Executive Blvd., EPS 7236, Rockville, MD 20892, USA. [email protected]

DOI: 10.1081/cnv-100103850

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https://www.tandfonline.com/doi/10.1081/cnv-100103...

MeSH Terms: Adenoma, Anti-Inflammatory Agents, Non-Steroidal, Apoptosis, Carcinoma, Cell Cycle Proteins, Cell Transformation, Neoplastic, Colorectal Neoplasms, Cyclin-Dependent Kinase Inhibitor p27, Dietary Fiber, Environmental Exposure, Enzyme Inhibitors, Folic Acid, Genes, APC, Genes, DCC, Genes, Tumor Suppressor, Genes, bcl-2, Genes, p53, Genes, ras, Humans, Methylation, Methylenetetrahydrofolate Reductase (NADPH2), Microtubule-Associated Proteins, Mutation, Oxidoreductases Acting on CH-NH Group Donors, Smoking, Tumor Suppressor Proteins

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